The Journal of clinical investigation
Endotoxin increases parathyroid hormone-related protein mRNA levels in mouse spleen. Mediation by tumor necrosis factor.
Chylomicrons alter the fate of endotoxin, decreasing tumor necrosis factor release and preventing death.
Cutaneous barrier perturbation stimulates cytokine production in the epidermis of mice.
Adenosine triphosphate stimulates phosphoinositide metabolism, mobilizes intracellular calcium, and inhibits terminal differentiation of human epidermal keratinocytes.
Role for monokines in the metabolic effects of endotoxin. Interferon-gamma restores responsiveness of C3H/HeJ mice in vivo.
Molecular and metabolic basis for the metabolic disorder normotriglyceridemic abetalipoproteinemia.
Expression of AT2 receptors in the developing rat fetus.
Familial chylomicronemia (type I hyperlipoproteinemia) due to a single missense mutation in the lipoprotein lipase gene.
Human very low density lipoproteins and chylomicrons can protect against endotoxin-induced death in mice.
Physiologic regulation of the serum concentration of 1,25-dihydroxyvitamin D by phosphorus in normal men.