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Endocrinology
Parathyroid hormone (PTH)/PTHrP receptor mutations in human chondrodysplasia.
Programmed Administration of Parathyroid Hormone Increases Bone Formation and Reduces Bone Loss in Hindlimb-Unloaded Ovariectomized Rats**This work was supported by NASA Grants NAGW-4963 and NCC-2–589. Additional support was obtained from NIH Grants AR-
Programmed administration of parathyroid hormone increases bone formation and reduces bone loss in hindlimb-unloaded ovariectomized rats.
Parathyroid hormone-related peptide delays terminal differentiation of chondrocytes during endochondral bone development.
Agonist-stimulated phosphorylation of the G protein-coupled receptor for parathyroid hormone (PTH) and PTH-related protein.
Keratinocyte growth factor increases fatty acid mobilization and hepatic triglyceride secretion in rats.
Endotoxin induces parathyroid hormone-related protein gene expression in splenic stromal and smooth muscle cells, not in splenic lymphocytes.
The molecular response of bone to growth hormone during skeletal unloading: regional differences.
The molecular response of bone to growth hormone during skeletal unloading: regional differences.
Interleukin-6 stimulates hepatic triglyceride secretion in rats.
